The anti-motility signaling mechanism of TGFβ3 that controls cell traffic during skin wound healing

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The anti-motility signaling mechanism of TGFβ3 that controls cell traffic during skin wound healing

When skin is wounded, migration of epidermal keratinocytes at the wound edge initiates within hours, whereas migration of dermal fibroblasts toward the wounded area remains undetectable until several days later. This "cell type traffic" regulation ensures proper healing of the wound, as disruptions of the regulation could either cause delay of wound healing or result in hypertrophic scars. TGFβ...

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ژورنال

عنوان ژورنال: Biology Open

سال: 2012

ISSN: 2046-6390

DOI: 10.1242/bio.20122246